Preliminary research reveal conolidine may perhaps inhibit certain ion channels, lessening neuronal excitability and limiting suffering indicators. This mechanism is particularly pertinent in neuropathic suffering, in which abnormal signaling brings about persistent discomfort. In addition, conolidine seems to affect G protein-coupled receptor (GPCR) pathways integral to ache notion. ... https://helenu681yvr1.wikicorrespondence.com/user